{"id":49544,"date":"2006-02-01T00:00:00","date_gmt":"2018-12-04T03:58:47","guid":{"rendered":"https:\/\/aprifel-pp.mentalworks.biz\/fr\/resume-scientifique\/cruciferous-vegetables-and-colo-rectal-cancer\/"},"modified":"2018-12-04T04:58:47","modified_gmt":"2018-12-04T03:58:47","slug":"cruciferous-vegetables-and-colo-rectal-cancer","status":"publish","type":"resume","link":"https:\/\/aprifel-pp.mentalworks.biz\/fr\/resume-scientifique\/cruciferous-vegetables-and-colo-rectal-cancer\/","title":{"rendered":"Cruciferous vegetables and colo-rectal cancer."},"content":{"rendered":"

Cruciferous vegetables have been studied extensively for their chemoprotective effects. Although they contain many bioactive compounds, the anti-carcinogenic actions of cruciferous vegetables are commonly attributed to their content of glucosinolates. Glucosinolates are relatively biologically inert but can be hydrolysed to a range of bioactive compounds such as isothiocyanates (ITC) and indoles by the plant-based enzyme myrosinase, or less efficiently by the colonic microflora. A number of mechanisms whereby ITC and indoles may protect against colo-rectal cancer have been identified. In experimental animals cruciferous vegetables have been shown to inhibit chemically-induced colon cancer. However, the results of recent epidemiological cohort studies have been inconsistent and this disparity may reflect a lack of sensitivity of such studies. Possible explanations for the failure of epidemiological studies to detect an effect include: assessment of cruciferous vegetable intake by methods that are subject to large measurement errors; the interaction between diet and genotype has not been considered: the effect that post-harvest treatments may have on biological effects of cruciferous vegetables has not been taken into account.<\/p>\n","protected":false},"template":"","mots_cles":[],"class_list":["post-49544","resume","type-resume","status-publish","hentry"],"acf":{"adresse":"The Robert Gordon University, Aberdeen, UK. 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